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The Melanin Scholars: Let's Debunk these Fools
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[QUOTE]Originally posted by michael1010: [QB] [QUOTE]Originally posted by Mike111: [qb] [b]THIS IA ACTUALLY A VERY NICE STUDY; TO STUDY A WHITE BOY TRYING TO BULLS1T HIMSELF AND ANYONE ELSE FOOLISH ENOUGH TO PAY ATTENTION! He is studying The Melanocortin 1 Receptor (MC1R): to find answers as to why White people are White. (He notes that when the MC1R mutates, it often causes a LOSS-of-FUNCTION in Humans. i.e. MC1R whole and UNCHANGED = GOOD. Mutated = BAD!).[/b] The Melanocortin 1 Receptor (MC1R): More Than Just Red Hair JONATHAN L. REES In man, a number of loss-of-function mutations in the MC1R have been described. The majority of red-heads (red-haired persons) are compound heterozygotes or homozygotes for up to five frequent loss-of-function mutations. A minority of red- heads are, however, only heterozygote. The MC1R is, there- fore, a major determinant of sun sensitivity and a genetic risk factor for melanoma and non-melanoma skin cancer. Recent work suggests that the MC1R also shows a clear heterozygote It is worth setting out the relevant questions Why are humans different in terms of skin colour and, more particularly, why is there such diversity of skin and hair colour in Northern Europeans? Is this natural selection favouring lightening of the skin or conversely is this merely due to absence of selection in Europe, with selection operat-ing against the development of MC1R mutants in Africa? [b][i](AS often happens, White boys will slip in a lie to start things off. i.e. There is VERY little diversity in SKIN color of Norther Europeans, the diversity is ONLY in HAIR colors: such as Red, Blond, Strawberry blond and ginger - ALL OF WHICH ARE SYMPTOMATIC OF ALBINISM!).[/i] [/b] How old are the various alleles and are these dates compat-ible with what we know of human migration out of Africa? Rana et al. (44) studied the MC1R in 121 individuals from population samples from around the world. They detected six MC1R variants and showed that the 163 variant as previously reported by Sturm's group (38) was common in Asian populations. Furthermore, they showed that the pattern of MC1R diversity was lower in African populations than in those from Northern Europe. They suggested that this could represent diversifying selection. More recently, my own group in collaboration with Rosalind Harding have published a larger study (42) involv-ing a more quantitative mathematical approach based on sequencing of 448 full sequences and have come to different conclusions (Fig. 1). Like Rana et al. we show that whilst there is considerably less diversity in African popula-tions than in European populations, comparison of the divergence between chimp and human sequence, together with allowance for selective ascertainment of functional mutations associated with red hair, suggests that the MC1R data is compatible with neutral theory in Europe but with functional constraint operating in Africa. In other words, [b]whilst the data provide evidence for selective pressure acting to maintain wild-type MC1R status in Africa,[/b] in Europe the data are compatible with a removal of constraint rather than selection in favour of loss-of-function mutants. [b]Did anyone catch the highlighted above. White boy is saying that a healthy MC1R is "WILD". Does that mean that the "Mutated", Fractured and Broken, UN-healthy European MC1R is CIVILIZED??? See xyyman, when it comes to their origins (Albinism), you can't pay attention to a thing they say - It's all going to be PURE Bullsh1t![/b] [/qb][/QUOTE]Stay away from the desktop you dumbass mofo. Everytime a child is born, they have a certain number of *mutated* genes, even if we don't know what function they serve. Being mutated don't have a goddamn thing to do with being *healthy*. I can't believe MelaninKing agreeing with your dumbass on this. I thought he was smarter. Both of you are some whack ass desktop scholars. [/QB][/QUOTE]
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